Doesn’t the “Law of Thermodynamics” Prove Eating Less Burns Body Fat?

Starvation Is NOT Healthy. Stop counting calories & go #SANE w/me at http://SANESolution.com

 

“The principle that weight gain [only depends on calorie quantity] would violate the second law of thermodynamics.” —R.D. Feinman, State University of New York

We know the traditional approach to fat loss fails 95% of the time, yet common sense seems to tell us: “If you eat less and exercise more, you must burn body fat. Anything else violates the law of thermodynamics.”

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There are four laws of thermodynamics. The two that apply to burning body fat do not prove that reducing the number of calories eaten makes the body burn fat. They tell us energy cannot be created nor destroyed; energy can only change forms. When people eat less, the body must do something. That’s it. The laws of thermodynamics prove nothingabout what the fat metabolism system must do.

Remember how it is easier for your body to slow down than to burn fat? And remember how it makes more sense to burn calorie-hungry muscle than it does to burn protective body fat? Put those two facts together, and instead of proving that eating less equals long-term fat loss, the applicable laws of thermodynamics prove that eating less makes the body slow down and burn muscle, which leads to long-term fat gain—not fat loss.


  1. Feinman RD, Fine EJ. “A calorie is a calorie” violates the second law of thermodynamics. Nutr J. 2004 Jul 28;3:9. PubMed PMID: 15282028; PubMed Central PMCID: PMC506782.
  2. Fine EJ, Feinman RD. Thermodynamics of weight loss diets. Nutr Metab (Lond).2004 Dec 8;1(1):15. PubMed PMID: 15588283; PubMed Central PMCID: PMC543577.
  3. Garrow, J.S.. Energy Balance and Obesity in Man. 2nd ed. New York: Elsevier Science Ltd, 1978. Print.
  4. Keesey RE, Powley TL. The regulation of body weight. Annu Rev Psychol. 1986;37:109-33. PubMed PMID: 3963779.
Learn the exact foods you must eat if you want to finally lose weight permanently. Click here to download your free Weight Loss Food List, the “Eat More, Lose More” Weight Loss Plan, and the “Slim in 6” Cheat Sheet…CLICK HERE FOR FREE “HOW TO” WEIGHT LOSS GUIDES

Q&A: Is SANE Eating the Same for Children and Adults?

Starvation Is NOT Healthy. Stop counting calories & go #SANE w/me at http://SANESolution.com

SANE Eating the Same for Children and Adults?

Yes and no. Yes, in that the same foods are SANE and inSANE for children and adults. No, in that SANE eating is more important for children because:

  1. Children require more nutrition than adults.
  2. Children are more susceptible to food-related behavior problems.
  3. Fat cells never go away.
  4. The habits children form affect them for the rest of their lives.

Children require more nutrition than adults

Most expectant mothers are especially careful about what they put into their bodies while they are pregnant. Why? Because they know optimal nutrition is critical for a developing fetus. Similarly, optimal nutrition is critical for a developing child.

SANE foods contain more nutrition per calorie than any other foods (The “N” in SANE stands for “Nutrition.”) When a child eats a SANE diet, that child is eating the most nutritious diet possible.

Learn the exact foods you must eat if you want to finally lose weight permanently. Click here to download your free Weight Loss Food List, the “Eat More, Lose More” Weight Loss Plan, and the “Slim in 6” Cheat Sheet…CLICK HERE FOR FREE “HOW TO” WEIGHT LOSS GUIDES

Children are more susceptible to food-related behavior problems

InSANE foods such as starches and sweets are dramatically more Aggressive (the “A” in SANE) than SANE foods. They release a significant and short burst of energy into the body. This causes a short energy high followed by longer-lasting lethargy. Still developing mentally and emotionally, children are doubly impacted by these “highs” and “lows,” which is why children start “bouncing off of the walls” and have a hard time concentrating after eating inSANE starches and sweets. It is also why children then become sluggish and have a hard time concentrating. SANE eating has long been “prescribed” to aid children said to be suffering from ADHD (attention deficit hyperactivity disorder).

A SANE diet ensures a slow and steady supply of energy to the body and enables optimal mood and behavior.

Fat cells never go away

A SANE diet has been proven to enable the body to burn rather than to store fat. Once fat cells are made, we cannot get rid of them; we can only shrink them. This is why helping our children avoid excess body fat is so important and why childhood obesity is so heartbreaking. Once a child develops new fat cells, that youngster will have a harder time staying slim for the rest of his or her life because those fat cells will never go away. At best, they will shrink, but still predispose that child to storing excess body fat.

The habits children form affect them for the rest of their lives

The habits we learn as children stick with us. When we teach our children SANE habits, we make it dramatically easier for them to keep themselves fit and healthy for the rest of their lives.

In summary, if it’s SANE or inSANE for an adult, it’s even more SANE or inSANE for a child. Everything that makes SANE eating important for adults makes it even more important for children.


  1. Ailhaud G, Hauner H. Development of White Adipose Tissue In: Bray GA, Couchard d, James WP, eds. Handbook of Obesity. New York: Marcel Dekker, 1997: 359-378.
  2. Faust IM, Johnson PR, Hirsch J. Surgical removal of adipose tissue alters feeding behavior and the development of obesity in rats. Science. 1977 Jul 22;197(4301):393-6. PubMed PMID: 877564.
  3. Krotkiewski M, Sjöström L, Björntorp P, Carlgren G, Garellick G, Smith U. Adipose tissue cellularity in relation to prognosis for weight reduction. Int J Obes. 1977;1(4):395-416. PubMed PMID: 617116.
  4. Obesity and leanness. Basic aspects. Stock, M., Rothwell, N., Author Affiliation: Dep. Physiology, St. George’s Hospital Medical School, London Univ., London, UK.
Learn the exact foods you must eat if you want to finally lose weight permanently. Click here to download your free Weight Loss Food List, the “Eat More, Lose More” Weight Loss Plan, and the “Slim in 6” Cheat Sheet…CLICK HERE FOR FREE “HOW TO” WEIGHT LOSS GUIDES

Exercising More Does Not Equal Long-Term Fat Loss

Starvation Is NOT Healthy. Stop counting calories & go #SANE w/me at http://SANESolution.com

 

“My grandmother, she started walking five miles a day when she was sixty. She’s ninety-seven today and we don’t know where the hell she is.” – Ellen DeGeneres

In the same way that people drink more fluids when they exercise more, they also eat more when they exercise more. Researcher Hugo R. Rony found: “Consistently high or low energy expenditures result in consistently high or low levels of appetite. Thus men doing heavy physical work spontaneously eat more than men engaged in sedentary occupations.” J.M. Friedman at Rockefeller University makes a similar point: “Exercise by itself has not been shown to be highly effective in treating obesity because the increased energy use from exercise is generally offset by increased caloric intake.”

Compounding the problem, many people who exercise more do not eat high-quality food. The majority of people get most of their calories from low-quality starches and sweeteners.Therefore, exercising more triggers the consumption of more low-quality food. More low-quality food means less need to burn body fat, more clogging, and a higher set-point. Far from burning body fat, we burn time and build-up clogs.

Learn the exact foods you must eat if you want to finally lose weight permanently. Click here to download your free Weight Loss Food List, the “Eat More, Lose More” Weight Loss Plan, and the “Slim in 6” Cheat Sheet…CLICK HERE FOR FREE “HOW TO” WEIGHT LOSS GUIDES

Here is one scenario for exercising more: Michelle goes for a 30-minute jog and burns 170 more calories than she would have burned by sitting at home and reading this book. She is trying hard to cut calories, so she does not drink any sugary sports drinks and fights through the hunger pangs after her jog. At dinner Michelle unconsciously drinks an extra glass of reduced-fat milk thanks to her increased thirst and hunger. The net result of her jog is thirteen more calories than if she had not exercised.

30 min. jog….….. -170 calories
12 oz. milk……… +183 calories
__________________________
Net………….….… +13 calories

Much more commonly, people will have sweetened “power juice” while pounding it out on the treadmill. Afterward, they overeat low-quality food. The net result is more low-quality food and more clogging.

30 min. jog………………………………………………………….…….-170 calories
24 oz. sports drink………………………………………..…………….+189 calories
Extra half serving of Fettuccine Alfredo………….…………………..+390 calories
_____________________________________________________________
Net…………………………………………………………………..……+409 calories

The food industry is very well aware that exercising more encourages eating more low-quality food. That’s why the following corporations serve on the executive board of the American Council on Fitness and Nutrition:

  • Coca-Cola Company
  • PepsiCo
  • Hershey Foods Corporation
  • Sara Lee Corporation
  • Kellogg Company
  • Kraft Foods
  • General Mills
  • Campbell Soup Company
  • ConAgra Foods
  • Del Monte Foods
  • Grocery Manufacturers Association
  • H.J. Heinz Company
  • Masterfoods USA
  • National Restaurant Association
  • Unilever United States
  • American Association of Advertising Agencies
  • American Beverage Association
  • Association of National Advertisers

Are we told to exercise more because it is good for fat loss or because it is good for business? The National Soft Drink Association advises us to “consume at least eight glasses of fluids daily, even more when you exercise. A variety of beverages, including soft drinks, can contribute to proper hydration.”

But wait. If you exercise less, won’t you gain body fat? As you’ll see in future posts that depends on the type of exercise you do. In the next two posts we’ll cover how exercising less doesnot cause long-term fat gain and eating more doesnot cause long-term fat gain.

Note: Being active is good for your health. We definitely should not sit around all day. Studies consistently show that physical activity boosts life expectancy. However, they do not show that traditional cardiovascular exercise effectively burns body fat long term.


  1. Cordain, Loren, and Joe Friel. The Paleo Diet for Athletes: A Nutritional Formula for Peak Athletic Performance. Emmaus, Pa.: Rodale Books, 2005. Print.
  2. Donnelly JE, Smith BK. Is exercise effective for weight loss with ad libitum diet? Energy balance, compensation, and gender differences. Exerc Sport Sci Rev. 2005 Oct;33(4):169-74. Review. PubMed PMID: 16239833.
  3. Friedman JM. Modern science versus the stigma of obesity. Nat Med. 2004 Jun;10(6):563-9. Review. PubMed PMID: 15170194.
  4. Koopmans HS. Internal signals cause large changes in food intake in one-way crossed intestines rats. Brain Res Bull. 1985 Jun;14(6):595-603. PubMed PMID: 3875383.
  5. Marion Nestle, quoted in : J.M. Hirsch, “Food Industry a Targe in Obesity Fight,” March 19, 2006. www.forbes.com/feeds/ap/2006/03/18/ap2605096.html.
  6. National Soft Drink Association. Soft Drinks: Balance, Variety, Moderation.
  7. Rony, Hugo R.. Obesity and Leanness. London, Great Britian : Lea & Febiger, 1940. Print.
  8. Simon, Michele. Appetite for Profit: How the food industry undermines our health and how to fight back. New York City, New York: Nation Books, 2006. Print.
  9. Feinstein AR. The treatment of obesity: an analysis of methods, results, and factors which influence success. J Chronic Dis. 1960 Apr;11:349-93. PubMed PMID: 13821960.
  10. Hu FB, Sigal RJ, Rich-Edwards JW, Colditz GA, Solomon CG, Willett WC, Speizer FE, Manson JE. Walking compared with vigorous physical activity and risk of type 2 diabetes in women: a prospective study. JAMA 1999;282:1433-1439.
  11. Hu FB, Willett WC, Li T, Stampfer MJ, Colditz GA, Manson JE. Adiposity as compared with physical activity in predicting mortality among women. N Engl J Med 2004;351:2694-2703.
  12. Johnson JL, Slentz CA, Houmard JA, Samsa GP, Duscha BD, Aiken LB, McCartney JS, Tanner CJ, Kraus WE. Exercise training amount and intensity effects on metabolic syndrome (from Studies of a Targeted Risk Reduction Intervention through Defined Exercise). Am J Cardiol. 2007 Dec 15;100(12):1759-66. Epub 2007 Oct 29. PubMed PMID: 18082522; PubMed Central PMCID: PMC2190779.
  13. Kannel WB, Wilson P, Blair SN. Epidemiological assessment of the role of physical activity and fitness in development of cardiovascular disease. Am Heart J 1985;109:876-885.
  14. Kesaniemi YK, Danforth E Jr, Jensen MD, Kopelman PG, Lefèbvre P, Reeder BA. Dose-response issues concerning physical activity and health: an evidence-based symposium. Med Sci Sports Exerc. 2001 Jun;33(6 Suppl):S351-8. PubMed PMID:11427759.
  15. Löllgen H, Böckenhoff A, Knapp G. Physical activity and all-cause mortality: an updated meta-analysis with different intensity categories. Int J Sports Med. 2009 Mar;30(3):213-24. Epub 2009 Feb 6. PubMed PMID: 19199202.
  16. Manson JE, Greenland P, LaCroix AZ, Stefanick ML, Mouton CP, Oberman A, Perri MG, Sheps DS, Pettinger MB, Siscovick DS. Walking compared with vigorous exercise for the prevention of cardiovascular events in women. N Engl J Med 2002;347:716-725.
  17. Manson JE, Hu FB, Rich-Edwards JW, Colditz GA, Stampfer MJ, Willett WC, Speizer FE, Hennekens CH.  A prospective study of walking as compared with vigorous exercise in the prevention of coronary heart disease in women. N Engl J Med 1999;341:650-658.
  18. Morris JN, Heady JA, Raffle PA, Roberts CG, Parks JW. Coronary heart-disease and physical activity of work. Lancet 1953;265:1111-1120.
  19. Oguma Y, Sesso HD, Paffenbarger RS Jr, Lee IM. Physical activity and all cause mortality in women: a review of the evidence. Br J Sports Med. 2002 Jun;36(3):162-72. Review. PubMed PMID: 12055109; PubMed Central PMCID: PMC1724493.
  20. Paffenbarger RS, Hale WE. Work activity and coronary heart mortality. N Engl J Med 1975;292:545-550.
  21. Prentice AM, Black AE, Coward WA, Cole TJ. Energy expenditure in overweight and obese adults in affluent societies: an analysis of 319 doubly-labelled water measurements. Eur J Clin Nutr. 1996 Feb;50(2):93-7. PubMed PMID: 8641251.
  22. Stofan JR, DiPietro L, Davis D, Kohl HW III, Blair SN. Physical activity patterns associated with cardiorespiratory fitness and reduced mortality: The Aerobics Center Longitudinal Study. Am J Public Health 1998;88:1807-1813.
  23. Wei M, Gibbons LW, Mitchell TL, Kampert JB, Lee CD, Blair SN. The association between cardiorespiratory fitness and impaired fasting glucose and type 2 diabetes mellitus in men. Ann Intern Med 1999;130:89-96.
  24. Wei M, Kampert JB, Barlow CE, Nichaman MZ, Gibbons LW, Paffenbarger RS Jr, Blair SN. Relationship between low cardiorespiratory fitness and mortality in normal-weight, overweight, and obese men. JAMA 1999;282:1547-1553.
  25. Blair SN, Kohl HW 3rd, Paffenbarger RS Jr, Clark DG, Cooper KH, Gibbons LW. Physical fitness and all-cause mortality. A prospective study of healthy men and women. JAMA. 1989 Nov 3;262(17):2395-401. PubMed PMID: 2795824.
Learn the exact foods you must eat if you want to finally lose weight permanently. Click here to download your free Weight Loss Food List, the “Eat More, Lose More” Weight Loss Plan, and the “Slim in 6” Cheat Sheet…CLICK HERE FOR FREE “HOW TO” WEIGHT LOSS GUIDES

Exercising Less Does Not Cause Long-Term Fat Gain

Starvation Is NOT Healthy. Stop counting calories & go #SANE w/me at http://SANESolution.com

 

“It is reasonable to assume that persons with relatively high daily energy expenditures would be less likely to gain weight over time compared with those who have low energy expenditures. So far, datato support this hypothesis are not particularly compelling.” – American Heart Association

The idea that we have an obesity epidemic because people are not exercising enough is a myth. Saffron A. Whitehead at St. George’s University of London reported: “Most studies show that the obese do about the same physical activity as [the] lean.”

Common sense tells us that if exercising less is the cause of our collective weight issues, we must be collectively exercising less. Are we?

Not even close.

Learn the exact foods you must eat if you want to finally lose weight permanently. Click here to download your free Weight Loss Food List, the “Eat More, Lose More” Weight Loss Plan, and the “Slim in 6” Cheat Sheet…CLICK HERE FOR FREE “HOW TO” WEIGHT LOSS GUIDES

The idea of aerobic exercise did not even exist in the mainstream until the 1968 publication of the book Aerobics by Dr. Kenneth H. Cooper. Dr. Entin, with the department of Biological Sciences at Northern Arizona University, explains the common view before then: “In the 1930’s and 40’s…high volume endurance training was thought to be bad for the heart. Through the ‘50’s and even ‘60’s, exercise was not thought to be useful…and endurance exercise was thought to be harmful to women.” During that same period the percent of obese Americans was dramatically lower than today. Nowadays, Americans exercise more than anyone else in the world and are the sixth heaviest population in the world. How could doing too little of something that we did even less of before the problem existed cause the problem?

Some people claim that we are getting heavier because we are using labor-saving devices. Yet that doesn’t make sense. The vast majority of labor-saving devices became common in households decades before obesity shot up. Use of dishwashers, washing machines, vacuum cleaners, and all the major labor-saving devices increased most between 1945 and 1965. However, obesity increased little during that time period. Use of these devices increased very little between 1978 and 1998 while obesity rates shot up. So how could labor-saving devices be the cause of weight problems?

Reread the quote from the American Heart Association at the start of this chapter. Digging into the data and abandoning assumptions about our activity levels, researchers like New York University’s Marion Nestle tell us, “…the activity levels of Americans appear to have changed little, if at all, from the 1970s to the 1990s.”

What about all the TV watching? That’s got to be the cause, right? That too does not correspond with the facts.  Tsinghua University professor Seth Roberts determined: “Time spent watching TV increased by 45% from 1965 to 1975, yet obesity increased little over that time; from 1975 to 1995, when obesity shot up, TV watching increased only a little.”

Eating lowerquality food creates the clog that causes chronic weight gain. People can be plenty active, and exercise for hours, but if they eat low-quality food, they will get clogged and gain body fat. Long-term weight gain is determined by food and exercise quality, not quantity.


  1. Haskell WL, Lee IM, Pate RR, Powell KE, Blair SN, Franklin BA, Macera CA, Heath GW, Thompson PD, Bauman A; American College of Sports Medicine; American Heart Association. Physical activity and public health: updated recommendation for adults from the American College of Sports Medicine and the American Heart Association. Circulation. 2007 Aug 28;116(9):1081-93. Epub 2007 Aug 1. PubMed PMID: 17671237.
  2. Whitehead, Saffron A.; Nussey, Stephen (2001). Endocrinology: an integrated approach. Oxford: BIOS. pp. 122. ISBN 1-85996-252-1.
  3. Dr. Pauline, Entin. “History of Exercise Science.” www2.nau.edu. Northern Arizona University, n.d. Web. 10 Feb. 2011. <http://jan.ucc.nau.edu/pe/exs190web/exs190history.htm>.
  4. http://apps.who.int/bmi/index.jsp
  5. Nestle M, Jacobson MF. Halting the obesity epidemic: a public health policy approach. Public Health Rep. 2000 Jan-Feb;115(1):12-24. PubMed PMID: 10968581; PubMed Central PMCID: PMC1308552.
  6. Roberts, Seth. The Shangri-La Diet: The No Hunger Eat Anything Weight-Loss Plan. Chicago: Perigee Trade, 2007. Print.
Learn the exact foods you must eat if you want to finally lose weight permanently. Click here to download your free Weight Loss Food List, the “Eat More, Lose More” Weight Loss Plan, and the “Slim in 6” Cheat Sheet…CLICK HERE FOR FREE “HOW TO” WEIGHT LOSS GUIDES

eat less, weigh more…for 118,801 folks, at least

Starvation Is NOT Healthy. Stop counting calories & go #SANE w/me at http://SANESolution.com

Half of going SANE is about eating more, smarter (the other half is exercising less, smarter).

This seems odd since we’ve all been told the more we eat the more we weigh. However, a quick stroll through the studies shows “more food = more fat” is a myth.

For example, Harvard researchers looked at a massive sample of 67,272 women and divided them into fifths according to the quantity of calories they ate. The general trend was the less ladies ate, the more they weighed.

The researchers then divided the women into fifths according to the quality of calories they ate. The lower the quality of their calories the more they weighed.


Learn the exact foods you must eat if you want to finally lose weight permanently. Click here to download your free Weight Loss Food List, the “Eat More, Lose More” Weight Loss Plan, and the “Slim in 6” Cheat Sheet…CLICK HERE FOR FREE “HOW TO” WEIGHT LOSS GUIDES

The cause of weight gain is too little quality, not too much quantity. And while we’re at it, let’s not forget the studies showing yo-yo dieting—the inevitable result of trying to eat less—increasing our risk of heart attack, stroke, diabetes, high blood pressure, cancer, immune system failure, eating disorders, impaired cognitive function, chronic fatigue, and depression. The results are in…studies show blindly eating less doesn’t make us thin. It makes us stocky, sick, and sad.

More surprising science.

The Harvard folks then took 51,529 men and divided them into fifths according to the quantity of calories they ate. The more folks ate the less they weighed.

Practical and permanent fat loss isn’t about cutting calories. It’s about intelligently increasing the quantity of high quality calories we eat. In the same Harvard study researchers divided the 51,529 guys into fifths according to the quality of calories they ate. The higher the quality of their calories the less they weighed.

The calorie quantity theory is fiction and fails because cutting calorie quantity fights against our basic biology. Our bodies don’t like starving. Crazy…I know. Studies show the only way to drop fat forever is to work with our bodies rather than to fight them. We do that by eating more–higher quality–calories. We eat more, smarter. And that works because a calorie isn’t a calorie…but more on that later.

Sampling of sources

  1. Blackburn GL, Wilson GT, Kanders BS, Stein LJ, Lavin PT, Adler J, Brownell KD. Weight cycling: the experience of human dieters. Am J Clin Nutr. 1989 May;49(5 Suppl):1105-9. PubMed PMID: 2718940.
  2. Blair SN, Shaten J, Brownell K, Collins G, Lissner L. Body weight change, all-cause mortality, and cause-specific mortality in the Multiple Risk Factor Intervention Trial. Ann Intern Med. 1993 Oct 1;119(7 Pt 2):749-57. PubMed PMID: 8363210.
  3. Bray GA. Obesity–a state of reduced sympathetic activity and normal or high adrenal activity (the autonomic and adrenal hypothesis revisited). Int J Obes. 1990;14 Suppl 3:77-91; discussion 91-2. Review. PubMed PMID: 2086518.
  4. Brownell KD, Rodin J. Medical, metabolic, and psychological effects of weight cycling. Arch Intern Med. 1994 Jun 27;154(12):1325-30. Review. PubMed PMID: 8002684.
  5. Green MW, Rogers PJ. Impaired cognitive functioning during spontaneous dieting. Psychol Med. 1995 Sep;25(5):1003-10. PubMed PMID: 8587997.
  6. Greene P, Willett W, et al. Pilot 12-week feeding weight loss comparison: low-fat vs. low-carbohydrate (ketogenic) diets [abstract]. Obes Res. 2003;11:A23.
  7. Hamm P, Shekelle RB, Stamler J. Large fluctuations in body weight during young adulthood and twenty-five-year risk of coronary death in men. Am J Epidemiol. 1989 Feb;129(2):312-8. PubMed PMID: 2912043.
  8. Higgins M, D’Agostino R, Kannel W, Cobb J, Pinsky J. Benefits and adverse effects of weight loss. Observations from the Framingham Study. Ann Intern Med. 1993 Oct 1;119(7 Pt 2):758-63. Erratum in: Ann Intern Med 1993 Nov 15;119(10):1055. PubMed PMID: 8363211.
  9. Hill AJ. Does dieting make you fat? Br J Nutr. 2004 Aug;92 Suppl 1:S15-8. Review. PubMed PMID: 15384316.
  10. Jeffery R. Prevention of Obesity. In: Bray GA, Couchard d, James WP, eds. Handbook of Obesity. New York: Marcel Dekker, 1997: 819-829.
  11. Keen H, Thomas BJ, Jarrett RJ, Fuller JH. Nutrient intake, adiposity, and diabetes. Br Med J. 1979 Mar 10;1(6164):655-8. PubMed PMID: 435710; PubMed Central PMCID: PMC1598272.
  12. Levine JA, Eberhardt NL, Jensen MD. Role of nonexercise activity thermogenesis in resistance to fat gain in humans. Science. 1999 Jan 8;283(5399):212-4. PubMed PMID: 9880251.
  13. Lissner L, Odell PM, D’Agostino RB, Stokes J 3rd, Kreger BE, Belanger AJ,Brownell KD. Variability of body weight and health outcomes in the Framingham population. N Engl J Med. 1991 Jun 27;324(26):1839-44. PubMed PMID: 2041550.
  14. Mann T, Tomiyama AJ, Westling E, Lew AM, Samuels B, Chatman J. Medicare’s search for effective obesity treatments: diets are not the answer. Am Psychol. 2007 Apr;62(3):220-33. Review. PubMed PMID: 17469900.
  15. McCullough ML, Feskanich D, Rimm EB, Giovannucci EL, Ascherio A, Variyam JN, Spiegelman D, Stampfer MJ, Willett WC. Adherence to the Dietary Guidelines for Americans and risk of major chronic disease in men. Am J Clin Nutr. 2000 Nov;72(5):1223-31. PubMed PMID: 11063453.
  16. McCullough ML, Feskanich D, Stampfer MJ, Rosner BA, Hu FB, Hunter DJ, Variyam JN, Colditz GA, Willett WC. Adherence to the Dietary Guidelines for Americans and risk of major chronic disease in women. Am J Clin Nutr. 2000 Nov;72(5):1214-22. PubMed PMID: 11063452.
  17. Phinney SD. Weight cycling and cardiovascular risk in obese men and women. Am J Clin Nutr. 1992 Oct;56(4):781-2. PubMed PMID: 1414977.
  18. Rolland-Cachera MF, Bellisle F. No correlation between adiposity and food intake: why are working class children fatter? Am J Clin Nutr. 1986 Dec;44(6):779-87. PubMed PMID: 3788830.
  19. Samaha FF, Iqbal N, Seshadri P, Chicano KL, Daily DA, McGrory J, Williams T, Williams M, Gracely EJ, Stern L. A low-carbohydrate as compared with a low-fat diet in severe obesity. N Engl J Med. 2003 May 22;348(21):2074-81. PubMed PMID: 12761364.
  20. Sondike, S., et al. “The Ketogenic Diet Increases Weight Loss But Not Cardiovascular Risk: A Randomized Controlled Trial.” Journal of Adolescent Health 26: 91, 2000
  21. Volek J, Sharman M, Gómez A, Judelson D, Rubin M, Watson G, Sokmen B, Silvestre R, French D, Kraemer W. Comparison of energy-restricted very low-carbohydrate and low-fat diets on weight loss and body composition in overweight men and women. Nutr Metab (Lond). 2004 Nov 8;1(1):13. PubMed PMID:15533250; PubMed Central PMCID: PMC538279.
  22. Weigle DS. Human obesity. Exploding the myths. West J Med. 1990 Oct;153(4):421-8. Review. PubMed PMID: 2244378; PubMed Central PMCID: PMC1002573.
  23. Wooley SC, Wooley OW, Dyrenforth S. The case against radical interventions. Am J Clin Nutr. 1980 Feb;33(2 Suppl):465-71. PubMed PMID: 7355820.
Learn the exact foods you must eat if you want to finally lose weight permanently. Click here to download your free Weight Loss Food List, the “Eat More, Lose More” Weight Loss Plan, and the “Slim in 6” Cheat Sheet…CLICK HERE FOR FREE “HOW TO” WEIGHT LOSS GUIDES

Want to Improve Your Cholesterol? Don’t Lower It. There’s a Smarter Approach.

Starvation Is NOT Healthy. Stop counting calories & go #SANE w/me at http://SANESolution.com

 

“There is a wealth of…evidence that increasing the concentration of HDL cholesterol through diet will lower the risk of coronary artery disease.” –R.P. Mensink, Maastricht University

In the last post we showed how Eating Fat Does Not Hurt Cholesterol & It’s Not About Lowering Cholesterol Anyway. Here’s where the confusion about cholesterol comes from in the first place. There are different types of cholesterol, and most of them are helpful or neutral. The two most commonly discussed are LDL (low-density lipoprotein) and HDL (high-density lipoprotein). They are required to produce new cells and hormones. Because of this critical role, even if we never ate any cholesterol, our liver or intestines would produce it.

What Are Healthy Cholesterol Numbers?

Table of Contents

When it comes to predicting heart health, the American Heart Association, International Diabetes Federation, and World Health Organization agree that low HDL cholesterol—not high LDL cholesterol—is what matters. And that low HDL is bad. Looking at disease and death rates at various levels of LDL and HDL cholesterol, researchers have found that people with low HDL run a much greater risk of heart disease.

 

Learn the exact foods you must eat if you want to finally lose weight permanently. Click here to download your free Weight Loss Food List, the “Eat More, Lose More” Weight Loss Plan, and the “Slim in 6” Cheat Sheet…CLICK HERE FOR FREE “HOW TO” WEIGHT LOSS GUIDES

Relative Risk of Heart Disease Given Total Cholesterol

(Total Cholesterol in Parenthesis)

 

There are two things to note about this graphic. First, total cholesterol is irrelevant. If someone tells you their total cholesterol is 185, what is their risk of heart disease? Looking at the preceding table, it is either very low or high, depending on how much of that 185 consists of HDL cholesterol. Similarly, if someone tells you their total cholesterol is 245, they either have a herculean heart or a hemorrhaging heart, depending on their HDL levels.

Second, note how increasing HDL cholesterol is more important for heart health than decreasing LDL cholesterol. High HDL cholesterol protects us from heart problems more than dropping our LDL levels ever could. Heart-healthy diets are not about lowering total cholesterol. They are about raising HDL cholesterol.

“…low HDL-cholesterol levels increase coronary heart disease risk…[programs] resulting in an increase in HDL-cholesterol levels could decrease the incidence of ischemic heart disease.” – J.P. Despres, Laval University

 

How to Raise HDL Cholesterol

The most effective way to raise our HDL levels is to eat more natural fat and less unnatural starch. Fat raises HDL. Starch lowers HDL.

The Impact of Fat and Starch on HDL and LDL Cholesterol and Health

 

A Smarter Science of Heart Healthy Foods

Since lower HDL does more harm than lower LDL does good, any diet which tells us to replace SANE sources of fat with inSANE starch worsens our cholesterol. This is why D. Mozaffarian at Harvard University wrote: “[Focusing] on effects of total and saturated fat on…total and low-density lipoprotein [LDL] cholesterol may have failed to reduce coronary heart disease risk and inadvertently worsened…insulin resistance, and weight gain.” Researcher A. Garg wrote the following in the Journal of the American Medical Association: “High-carbohydrate diets…caused persistent deterioration of glycemic control and accentuation of hyperinsulinemia [caused clogs], as well as increased…very-low-density lipoprotein [bad] cholesterol levels.”

Regrettably, under the government’s guidelines, we are supposed to replace natural foods containing fat with low-fat-high-starch products to lower our total cholesterol. Why? Lower total cholesterol is meaningless, and lower HDL cholesterol is terrible for us. Researchers have demonstrated this for decades.

For example, the February 1989 issue of the Diabetes Care journal put out by the American Diabetes Association contained a study comparing the government’s diet with a more SANE way of eating. The study concluded: “VLDL [bad] cholesterol was significantly increased…High-density lipoprotein [good] cholesterol concentrations were significantly decreased after consumption of the 60% carbohydrate diet.”

Comparable results were found with the equally imbalanced U.K. dietary guidelines. In the words of University of Glasgow researcher S.R. Arefhosseini: “Following the U.K. dietary guidelines resulted in changes…more likely to favor an increased risk of coronary heart disease.”

 

What About Saturated Fat and Cholesterol?

Even saturated fats are not cholesterol criminals. The American Heart Association found:

“No adequately designed randomized controlled study in the general population has shown that…decreasing saturated fat…intake significantly decreases coronary heart disease mortality.”

 

Bottom Line: Ways to Improve Cholesterol

What is the bottom line? Studies show that any diet telling you to replace SANE sources of fat with inSANE starches is unhealthy and fattening. M.L. McCullough at Harvard University made this point: “Limiting unsaturated fats, which is usually done by increasing carbohydrates…is detrimental…. Low-fat, high-carbohydrate diets provide a higher glycemic load, aggravate hyperinsulinemia [clogging], and may thus increase the risk of diabetes and coronary artery disease.”

Want to improve your cholesterol naturally? Eat more, but smarter.

Sadly, the government’s diet tells us to do exactly what science says we should avoid.

If you think this is troubling, wait until you see what happened when big business jumped on the government bandwagon. We’ll cover that next week.


  1. “Cholesterol : LIPID MAPS–Nature Lipidomics Gateway.” Home : LIPID MAPS–Nature Lipidomics Gateway. N.p., n.d. Web. 3 Jan. 2011. <http://www.lipidmaps.org/update/2009/090501/full/lipidmaps.2009.3.html>.
  2. Apoundert CM, Campos H, Stampfer MJ, et al. Blood levels of long-chain n-3 fatty acids and the risk of sudden death. N Engl J Med 2002;346:1113-1118.
  3. Apoundert CM, Gaziano JM, Willett WC, Manson JE. Nut consumption and decreased risk of sudden cardiac death in the Physicians’ Health Study. Arch Intern Med. 2002;162:1382-1387.
  4. Arefhosseini SR, Edwards CA, Malkova D, Higgins S. Effect of advice to increase carbohydrate and reduce fat intake on dietary profile and plasma lipid concentrations in healthy postmenopausal women. Ann Nutr Metab. 2009;54(2):138-44. Epub 2009 Apr 1. PubMed PMID: 19339775.
  5. Augustin LS, Franceschi S, Jenkins DJ, Kendall CW, La Vecchia C. Glycemic index in chronic disease: a review. Eur J Clin Nutr. 2002 Nov;56(11):1049-71. Review. PubMed PMID: 12428171.
  6. Coulston AM, Hollenbeck CB, Swislocki AL, Reaven GM. Persistence of hypertriglyceridemic effect of low-fat high-carbohydrate diets in NIDDM patients. Diabetes Care. 1989 Feb;12(2):94-101. PubMed PMID: 2539286.
  7. Despres JP. Krauss R. Obesity and Lipoprotein Metabolism In: Bray GA, Couchard d, James WP, eds. Handbook of Obesity. New York: Marcel Dekker, 1997: 651-675.
  8. Gardner CD, Kiazand A, Alhassan S, Kim S, Stafford RS, Balise RR, Kraemer HC, King AC. Comparison of the Atkins, Zone, Ornish, and LEARN diets for change in weight and related risk factors among overweight premenopausal women: The A TO Z Weight Loss Study: a randomized trial. JAMA. 2007 Mar 7;297(9):969-77. Erratum in: JAMA. 2007 Jul 11;298(2):178. PubMed PMID: 17341711.
  9. German JB, Dillard CJ. Saturated fats: what dietary intake? Am J Clin Nutr. 2004 Sep;80(3):550-9. Review. PubMed PMID: 15321792.
  10. Grundy SM, Cleeman JI, Daniels SR, Donato KA, Eckel RH, Franklin BA, Gordon DJ, Krauss RM, Savage PJ, Smith SC, Jr, Spertus JA Costa F. Diagnosis and Management of the Metabolic Syndrome: An American Heart Association/National Heart, Lung, and Blood Institute Scientific Statement. Circulation 2005, 112:2735-2752: originally published online September 12, 2005; doi: 10.1161/CIRCULATIONAHA.105.169404]
  11. Haskell WL, Lee IM, Pate RR, Powell KE, Blair SN, Franklin BA, Macera CA, Heath GW, Thompson PD, Bauman A; American College of Sports Medicine; American Heart Association. Physical activity and public health: updated recommendation for adults from the American College of Sports Medicine and the American Heart Association. Circulation. 2007 Aug 28;116(9):1081-93. Epub 2007 Aug 1. PubMed PMID: 17671237.
  12. http://www.idf.org/webdata/docs/IDF_Meta_def_final.pdf
  13. Hu FB, Manson JE, Willett WC. Types of dietary fat and risk of coronary heart disease: a critical review. J Am Coll Nutr. 2001 Feb;20(1):5-19. Review. PubMed PMID: 11293467.
  14. Hu FB, Stampfer MJ, Manson JE, et al. Dietary fat intake and the risk of coronary heart disease in women. N Engl J Med 1997;337:1491–9.
  15. Jump DB, Clarke SD, thelen A, Liimatta M. Coordinate regulation of glycolytic and lipogenic gene expression by polyunsaturated fatty acids. J Lipid Res. 1994 Jun;35(6):1076-84. PubMed PMID: 8077846.
  16. Kersten S. Mechanisms of nutritional and hormonal regulation of lipogenesis. EMBO Rep. 2001 Apr;2(4):282-6. Review. PubMed PMID: 11306547; PubMed Central PMCID: PMC1083868.
  17. Lee-Han H, Cousins M, Beaton M, McGuire V, Kriukov V, Chipman M, Boyd N. Compliance in a randomized clinical trial of dietary fat reduction in patientswith breast dysplasia. Am J Clin Nutr. 1988 Sep;48(3):575-86. PubMed PMID: 3046298.
  18. Lopez-Garcia E, Schulze MB, Manson JE, Meigs JB, Apoundert CM, Rifai N, Willett WC, Hu FB. Consumption of (n-3) fatty acids is related to plasma biomarkers of inflammation and endothelial activation in women. J Nutr. 2004 Jul;134(7):1806-11. PubMed PMID: 15226473.
  19. Marchioli R, Barzi F, Bomba E, et al, GISSI-Prevenzione Investigators. Early protection against sudden dealth by n-3 polyunsaturated fatty acids after myocardial infarction: Time-course analysis of the results of the Gruppo Italiano per lo Studio della Sopravvivenza nell’Infarto Miocardico (GISSI)-Prevenzione. Circulation. 2002;105:1897-1903.
  20. McCullough ML, Feskanich D, Stampfer MJ, Rosner BA, Hu FB, Hunter DJ, Variyam JN, Colditz GA, Willett WC. Adherence to the Dietary Guidelines for Americans and risk of major chronic disease in women. Am J Clin Nutr. 2000 Nov;72(5):1214-22. PubMed PMID: 11063452.
  21. Mensink RP, Katan MB. Effect of monounsaturated fatty acids versus complex carbohydrates on high-density lipoproteins in healthy men and women. Lancet. 1987 Jan 17;1(8525):122-5. PubMed PMID: 2879969.
  22. Mensink RP, Zock PL, Kester AD, Katan MB. Effects of dietary fatty acids and carbohydrates on the ratio of serum total to HDL cholesterol and on serum lipids and apolipoproteins: a meta-analysis of 60 controlled trials. Am J Clin Nutr. 2003 May;77(5):1146-55. PubMed PMID: 12716665.
  23. Mensink RP, Zock PL, Kester AD, Katan MB. Effects of dietary fatty acids and carbohydrates on the ratio of serum total to HDL cholesterol and on serum lipids
  24. Mensink RPM, Katan MB. Effect of dietary trans fatty acids on highdensity and low-density lipoprotein cholesterol levels in healthy subjects. N Engl J Med 1990;323:439–45.
  25. Mori TA, Beilin LJ. Omega-3 fatty acids and inflammation. Curr Atheroscler Rep. 2004 Nov;6(6):461-7. Review. PubMed PMID: 15485592.
  26. Mozaffarian D. Effects of dietary fats versus carbohydrates on coronary heart disease: a review of the evidence. Curr Atheroscler Rep. 2005 Nov;7(6):435-45.Review. PubMed PMID: 16256001     &     Garg A, Bantle JP, Henry RR, Coulston AM, Griver KA, Raatz SK, Brinkley L, Chen YD, Grundy SM, Huet BA, et al. Effects of varying carbohydrate content of diet in patients with non-insulin-dependent diabetes mellitus. JAMA. 1994 May 11;271(18):1421-8. PubMed PMID: 7848401.
  27. Parks EJ, Hellerstein MK. Carbohydrate-induced hypertriacylglycerolemia: historical perspective and review of biological mechanisms. Am J Clin Nutr. 2000 Feb;71(2):412-33. Review. PubMed PMID: 10648253.
  28. Pirozzo S, Summerbell C, Cameron C, Glasziou P. Should we recommend low-fat diets for obesity? Obes Rev. 2003 May;4(2):83-90. Review. Erratum in: Obes Rev. 2003 Aug;4(3):185. PubMed PMID: 12760443.
  29. Ravnskov U. The questionable role of saturated and polyunsaturated fatty acids in cardiovascular disease. J Clin Epidemiol. 1998 Jun;51(6):443-60. Review. PubMed PMID: 9635993.
  30. Sacks FM, Katan M. Randomized clinical trials on the effects of dietary fat and carbohydrate on plasma lipoproteins and cardiovascular disease. Am J Med. 2002 Dec 30;113 Suppl 9B:13S-24S. Review. PubMed PMID: 12566134.
  31. Sands SA, Reid KJ, Windsor SL, Harris WS. The impact of age, body mass index, and seafood intake on the EPA and DHA content of human erythrocytes. Lipids 2005;40:343-347.
  32. Sharman MJ, Kraemer WJ, Love DM, Avery NG, Gómez AL, Scheett TP, Volek JS. A ketogenic diet favorably affects serum biomarkers for cardiovascular disease in normal-weight men. J Nutr. 2002 Jul;132(7):1879-85. PubMed PMID: 12097663.
  33. Simopoulos AP. Essential fatty acids in health and chronic disease. Am J Clin  Nutr. 1999 Sep;70(3 Suppl):560S-569S. Review. PubMed PMID: 10479232.
  34. Simopoulos AP. The importance of the ratio of omega-6/omega-3 essential fatty acids. Biomed Pharmacother. 2002 Oct;56(8):365-79. Review. PubMed PMID: 12442909.
  35. Siscovick DS, Raghunathan TE, King I, Weinmann S, Wicklund KG, Apoundright J, Bovbjerg V, Arbogast P, Smith H, Kushi LH, Cobb LA, Copass MK, Psaty BM, Lemaitre R, Retzlaff B, Childs M, Knopp RH. Dietary intake and cell membrane levels of long-chain n-3 polyunsaturated fatty acids and the risk of primary cardiac arrest. JAMA 1995;274:1363-1367.
  36. Willett W. Challenges for public health nutrition in the 1990s. Am J Public Health. 1990 Nov;80(11):1295-8. PubMed PMID: 2240291; PubMed Central PMCID: PMC1404889.
  37. Willett WC, Stampfer MJ, Manson JE, et al. Intake of trans fatty acids and risk of coronary heart disease among women. Lancet 1993;341:581–5.
  38. Wood, Philip A.. How fat works. Cambridge: Harvard University Press, 2006. Print.
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